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FOREWORD – HEART INTERNATIONAL – VOLUME 19 ISSUE 1 – 2025

Magdi El-Omar

Cardiovascular medicine stands at a pivotal crossroads—shaped by rapid advances in precision therapies, a deepening understanding of disease mechanisms, and an urgent imperative to address global health disparities. As the burden of cardiovascular disease continues to evolve, so too does the need for nuanced, evidence-based approaches that span the full spectrum of care: from prevention […]

Heart International. 2025;19(1):1-2

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HEART INTERNATIONAL VOLUME 6 ISSUE 1

Arrhythmia Imaging

Pseudo-dissection of ascending aorta in inferior myocardial infarction

< 1 min

Heart Failure

Malnutrition as assessed by nutritional risk index is associated with worse outcome in patients admitted with acute decompensated heart failure: an ACAP-HF data analysis

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Interventional Cardiology

Pressure ulcer: an unreported complication of the Safeguard® hemostasis device. No need to crack under pressure

< 1 min

Congenital Conditions

Association of interatrial septal abnormalities with cardiac impulse conduction disorders in adult patients: experience from a tertiary center in Kosovo

< 1 min

Atherosclerosis Imaging

Single coronary artery from the right sinus of Valsalva

< 1 min

Congenital Conditions

Accessory mitral valve tissue causing severe left ventricular outflow tract obstruction in a post-Senning patient with transposition of the great arteries

< 1 min

Arrhythmia

Implications of turbulence slope variations in different approaches

< 1 min

Cardiovascular Disease

Does high-dose metformin cause lactic acidosis in type 2 diabetic patients after CABG surgery? A double blind randomized clinical trial

< 1 min

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Monitoring Disease Progression in Transthyretin Amyloid Cardiomyopathy

Adam Ioannou

Transthyretin amyloid cardiomyopathy (ATTR-CM) is an inexorably progressive and ultimately fatal cardiomyopathy characterized by the deposition of misfolded transthyretin (TTR) in the form of amyloid fibrils within the myocardium.1 Transthyretin amyloid (ATTR) fibrils are insoluble protease–resistant beta-pleated sheets that are stabilized within the extracellular matrix, disrupting the integrity, structure and function of the myocardium.2 ATTR-amyloid fibril […]

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